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The triagonist displayed balanced subnanomolar potency at all three human receptors in albumin-bound conditions, with picomolar incretin activity and low nanomolar FGF21 activity

In rat tendon studies, administration accelerated outgrowth, fibroblast migration, and post-injury cellular survival (PMID 21030672)

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The complex reciprocal reinforcement between metabolic and immunological pathways makes MASH an immunometabolic disease

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